Gastrointestinal (GI): NSAIDs that block COX-one activity have correlations with GI problems due to inhibition with the synthesis of gastroprotective agents including PGE2 and also other prostaglandins. This inhibition results in diminished mucin creation by gastric epithelial cells, a lot less bicarbonate secretion, and fewer epithelial cell turnover, amongst other https://robertof538ttq5.ourcodeblog.com/profile