Max in WT synaptoneurosomes, suggesting that Src signaling could be downregulated in KI synapses. Alternatively, our capacity to rescue SERT purpose in KI midbrain synaptoneurosomes because of the inhibition of FAK suggests elevated FAK signaling downstream on the Pro32Pro33 mutant, as confirmed by improved pFAK localization in 5-HT synapses. Our https://rylanpdqal.blogchaat.com/32300191/pro33-an-overview