Preliminary research show conolidine might inhibit unique ion channels, minimizing neuronal excitability and limiting ache indicators. This mechanism is especially relevant in neuropathic soreness, in which irregular signaling brings about persistent discomfort. Also, conolidine seems to impact G protein-coupled receptor (GPCR) pathways integral to suffering notion. Computational dock... https://fredb913apf4.newsbloger.com/profile